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The following represent thyroid-related publications from all ATA-supported grant recipients, arranged by the year the award was made, covering the five year period following the award.
2002
Antonio C. Bianco, MD, PhD-Brigham and Women's Hospital/Harvard
Medical School
“Type 2 deiodinase-mediated intra-cellular thyrotoxicosis in brown
adipocytes is critical for energy homeostasis and adaptive thermogenesis
in small mammals”
1. Nakayama A, Bianco AC, Zhang CY, Lowell BB, Frangioni JV 2003. Quantitation of brown adipose tissue perfusion in transgenic mice using near-infrared fluorescence imaging. Mol Imaging 2:37-49.
2. Baqui M, Botero D, Gereben B, Curcio C, Harney JW, Salvatore D, Sorimachi K, Larsen PR, Bianco AC 2003. Human type 3 iodothyronine selenodeiodinase is located in the plasma membrane and undergoes rapid internalization to endosomes. J Biol Chem 278:1206-1211.
3. Curcio-Morelli C, Zavacki AM, Christofollete M, Gereben B, de Freitas BC, Harney JW, Li Z, Wu G, Bianco AC 2003. Deubiquitination of type 2 iodothyronine deiodinase by von Hippel-Lindau protein-interacting deubiquitinating enzymes regulates thyroid hormone activation. J Clin Invest 112:189-196.
4. Freitas FR, Moriscot AS, Jorgetti V, Soares AG, Passarelli M, Scanlan TS, Brent GA, Bianco AC, Gouveia CH 2003. Spared bone mass in rats treated with thyroid hormone receptor TR beta-selective compound GC-1. Am J Physiol Endocrinol Metab 285:E1135-E1141.
5. Kim BW, Zavacki AM, Curcio-Morelli C, Dentice M, Harney JW, Larsen PR, Bianco AC 2003. Endoplasmic reticulum-associated degradation of the human type 2 iodothyronine deiodinase (D2) is mediated via an association between mammalian UBC7 and the carboxyl region of D2. Mol Endocrinol 17:2603-2612.
6. Christoffolete MA, Linardi CC, de Jesus L, Ebina KN, Carvalho SD, Ribeiro MO, Rabelo R, Curcio C, Martins L, Kimura ET, Bianco AC 2004. Mice with targeted disruption of the Dio2 gene have cold-induced overexpression of the uncoupling protein 1 gene but fail to increase brown adipose tissue lipogenesis and adaptive thermogenesis. Diabetes 53:577-584.
Peter Kopp, MD-Northwestern University
“Targeted overexpression of a dominant negative insulin growth
factor I (IGFI) in thyroid cells”
1. Kopp P 2002. Perspective: genetic defects in the etiology of congenital hypothyroidism. Endocrinology 143:2019-2024.
2. Gillam MP, Sidhaye AR, Lee EJ, Rutishauser J, Stephan CW,
Kopp P 2004. Functional characterization of pendrin in a polarized
cell system. Evidence for pendrin-mediated apical iodide efflux. J Biol
Chem 279:13004-13010.
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